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Innovations in this field are fairly recent. The first functioning genetronic replicator was designed and constructed by Doctor Toby Russel in the mid-24th century. Dr. Russel's theory was that the device could scan a person's DNA and damaged organs, then using this information to replicate a new healthy organ. The first recipient to survive the use of this technology was a Klingon male, Worf, in 2368. He was struck by a falling container and, as a result, was partially paralyzed when the container broke his spine. In the case of the Klingon male, Dr. Russel proposed the replication of a new spinal column to give the male full mobility back. The patient nearly died during the operation, but survived due to redundancies in his biological systems. The process was later refined by Doctor Simon Tarses when it was used in combination with nanotechnology to repair the damaged portions of a Bajoran female's spine when it was severed by Taran'atar's attack (Memory Beta, 2394). | Innovations in this field are fairly recent. The first functioning genetronic replicator was designed and constructed by Doctor Toby Russel in the mid-24th century. Dr. Russel's theory was that the device could scan a person's DNA and damaged organs, then using this information to replicate a new healthy organ. The first recipient to survive the use of this technology was a Klingon male, Worf, in 2368. He was struck by a falling container and, as a result, was partially paralyzed when the container broke his spine. In the case of the Klingon male, Dr. Russel proposed the replication of a new spinal column to give the male full mobility back. The patient nearly died during the operation, but survived due to redundancies in his biological systems. The process was later refined by Doctor Simon Tarses when it was used in combination with nanotechnology to repair the damaged portions of a Bajoran female's spine when it was severed by Taran'atar's attack (Memory Beta, 2394). | ||
[[File:Laelspinalinjury_postsurgery.jpeg|100px|left|thumb|Lael's spinal injury after surgery.]] | |||
<h3>Conceptual Framework</h3> | <h3>Conceptual Framework</h3> | ||
The patient suffered broken lower lumbar vertabrae and nerve damage as a result of her fall. Though doctors<br />were able to reconstruct most of her damaged spinal column, the fragile nature of the vertabrae combined with the severe nerve damage offered no hope that the patient would be able to regain her mobility. This was only achieved by the introduction of specifically-programmed nanites which were able to reverse the nerve damage and strengthen the vertabrae. However, the nature of the injury resulted quicker-than-usual degradation of nerve fibers and already-weak bone. Despite daily injections of nanites with the intention of slowing the damage, all attempts have proven to do so only temporarily. It was estimated that without drastic action, the patient would lose mobility completely by age 30. | |||
As stated previously, factors that could impact the result of the procedures proposed in the methods section include the patient's hybrid physiology, age, gender, diet and exercise routines, and the unique nature of the patient's injuries. Another factor that could have a radical effect is the specific programming of the nanites currently in the patient's body. As with multiple sclerosis, there is concern that the nanites' attempts to repair damage to the nerve fibers is moot in that the myelin sheath surrounding these fibers is degrading to a point that messages to and from the brain are being slowed, resulting in decreased mobility. | As stated previously, factors that could impact the result of the procedures proposed in the methods section include the patient's hybrid physiology, age, gender, diet and exercise routines, and the unique nature of the patient's injuries. Another factor that could have a radical effect is the specific programming of the nanites currently in the patient's body. As with multiple sclerosis, there is concern that the nanites' attempts to repair damage to the nerve fibers is moot in that the myelin sheath surrounding these fibers is degrading to a point that messages to and from the brain are being slowed, resulting in decreased mobility. |